Systemic lupus erythematosus (SLE) is an inflammatory disease of unknown cause, characterized biologically by producing multiple autoantibodies, the most characteristics are directed against some kernel components such as deoxyribonucleic acid and native nucleosomes. Neurological manifestations are frequent and polymorphous. They are dominated by central attacks, while peripheral attacks are rarer. We report the case of LS, 34 years old, female, Senegalese, divorced, and followed for years for a SLE, living in Dakar, hospitalized in October 2016 at the Neurological Clinic of FANN National Teaching Hospital, Dakar-Senegal for a demyelinating polyneuropathy complicating Systemic lupus erythematosus (SLE). An electro neuro myogram showed elongation of distal latencies in the lower limbs, lengthening of F waves in the lower limbs, and decreased conduction velocity in the lower limbs. The search for native anti-DNA antibodies was positive. The outcome was fatal with one death on day 41 of his hospitalization.
Published in | Clinical Neurology and Neuroscience (Volume 1, Issue 4) |
DOI | 10.11648/j.cnn.20170104.12 |
Page(s) | 80-83 |
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This is an Open Access article, distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution and reproduction in any medium or format, provided the original work is properly cited. |
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Copyright © The Author(s), 2017. Published by Science Publishing Group |
Systemic Lupus Erythematosus, Polyneuropathy, Demyelinating
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APA Style
Patrice Ntenga, K. Kabulo, Bugeme M., Mourabit S., S. Boubacar, et al. (2017). Demyelinating Polyneuropathy Complicating Systemic Lupus Erythematosus: A Case Report. Clinical Neurology and Neuroscience, 1(4), 80-83. https://doi.org/10.11648/j.cnn.20170104.12
ACS Style
Patrice Ntenga; K. Kabulo; Bugeme M.; Mourabit S.; S. Boubacar, et al. Demyelinating Polyneuropathy Complicating Systemic Lupus Erythematosus: A Case Report. Clin. Neurol. Neurosci. 2017, 1(4), 80-83. doi: 10.11648/j.cnn.20170104.12
@article{10.11648/j.cnn.20170104.12, author = {Patrice Ntenga and K. Kabulo and Bugeme M. and Mourabit S. and S. Boubacar and A. Nahantchi and Mansare L. M. and Cisse O. and Bakhroum M. and Touré K. and M. Ndiaye and M. M. Ndiaye}, title = {Demyelinating Polyneuropathy Complicating Systemic Lupus Erythematosus: A Case Report}, journal = {Clinical Neurology and Neuroscience}, volume = {1}, number = {4}, pages = {80-83}, doi = {10.11648/j.cnn.20170104.12}, url = {https://doi.org/10.11648/j.cnn.20170104.12}, eprint = {https://article.sciencepublishinggroup.com/pdf/10.11648.j.cnn.20170104.12}, abstract = {Systemic lupus erythematosus (SLE) is an inflammatory disease of unknown cause, characterized biologically by producing multiple autoantibodies, the most characteristics are directed against some kernel components such as deoxyribonucleic acid and native nucleosomes. Neurological manifestations are frequent and polymorphous. They are dominated by central attacks, while peripheral attacks are rarer. We report the case of LS, 34 years old, female, Senegalese, divorced, and followed for years for a SLE, living in Dakar, hospitalized in October 2016 at the Neurological Clinic of FANN National Teaching Hospital, Dakar-Senegal for a demyelinating polyneuropathy complicating Systemic lupus erythematosus (SLE). An electro neuro myogram showed elongation of distal latencies in the lower limbs, lengthening of F waves in the lower limbs, and decreased conduction velocity in the lower limbs. The search for native anti-DNA antibodies was positive. The outcome was fatal with one death on day 41 of his hospitalization.}, year = {2017} }
TY - JOUR T1 - Demyelinating Polyneuropathy Complicating Systemic Lupus Erythematosus: A Case Report AU - Patrice Ntenga AU - K. Kabulo AU - Bugeme M. AU - Mourabit S. AU - S. Boubacar AU - A. Nahantchi AU - Mansare L. M. AU - Cisse O. AU - Bakhroum M. AU - Touré K. AU - M. Ndiaye AU - M. M. Ndiaye Y1 - 2017/08/07 PY - 2017 N1 - https://doi.org/10.11648/j.cnn.20170104.12 DO - 10.11648/j.cnn.20170104.12 T2 - Clinical Neurology and Neuroscience JF - Clinical Neurology and Neuroscience JO - Clinical Neurology and Neuroscience SP - 80 EP - 83 PB - Science Publishing Group SN - 2578-8930 UR - https://doi.org/10.11648/j.cnn.20170104.12 AB - Systemic lupus erythematosus (SLE) is an inflammatory disease of unknown cause, characterized biologically by producing multiple autoantibodies, the most characteristics are directed against some kernel components such as deoxyribonucleic acid and native nucleosomes. Neurological manifestations are frequent and polymorphous. They are dominated by central attacks, while peripheral attacks are rarer. We report the case of LS, 34 years old, female, Senegalese, divorced, and followed for years for a SLE, living in Dakar, hospitalized in October 2016 at the Neurological Clinic of FANN National Teaching Hospital, Dakar-Senegal for a demyelinating polyneuropathy complicating Systemic lupus erythematosus (SLE). An electro neuro myogram showed elongation of distal latencies in the lower limbs, lengthening of F waves in the lower limbs, and decreased conduction velocity in the lower limbs. The search for native anti-DNA antibodies was positive. The outcome was fatal with one death on day 41 of his hospitalization. VL - 1 IS - 4 ER -